ich on tang (new uv sterilizer)

And possibly cause liver damage, as fish can't process it properly:


Metabolic and histological studies on the effect of garlic administration on the carnivorous fish Chrysichthys auratus
Mohamed B. Al-Salahy, Abd Allah B. Mahmoud

Abstract

Chrysichthys auratus fish were orally administered with doses of garlic (Allium sativum) juice equivalent to 2g per kg body wt, as a single dose or the same dose every 48h for 5 and 11 days. The results showed a hypoglycaemia, hypolipidaemia, hypocholesterolaemia, hypotriglyceridaemia and drop in serum TFAA as well as promotion of lipogenesis and glycogenesis in white muscle in response to repeated doses of garlic. Such results may correlate with elevation of insulin release in the fish following garlic administration. Histological and biochemical studies confirmed liver glycogen depletion and liver histopathological changes including vacuolation, hypertrophy and degeneration of some hepatocytes, and dilatation and congestion of some blood vessels. Histopathological changes of the kidney were vacuolation and rupture of some renal tubules, and degeneration of the parietal cells of some Malpighian corpuscles. Garlic decreased the amount of collagenous fibres in both liver and kidney, and reduced carbohydrate content in the kidney. In spite of some cell degeneration in the liver and renal tubules, serum AST and ALT did not show any significant changes, while serum ALP was decreased. Generally, it could be concluded that garlic strongly affected carbohydrate metabolism in Chrysichthys auratus. Garlic may improve carbohydrate metabolism, probably lowering the dietary protein costs of the fish; the damage of garlic on both liver and kidney in this work may be attributed to the relative high dose of garlic used.



Some physiological studies on the effect of onion and garlic juices on the fish, Clarias lazera

M.B. Al-Salahy

Abstract
Fish, Clarias Lazera were orally administered onion (Allium cepa) or garlic (Allium sativum) juices. Contents of glucose, free amino acids, total lipids, triglycerides, cholesterol, albumin, total protein, urea, aspartate aminotransferase (AST) and alanine aminotransferase (ALT) in serum as well as glycogen, free amino acids, total lipids, triglycerides and cholesterol contents in liver, white and red muscle were determined 5 h and 24 h after a single dose or after repeated doses (1 dose every 24 h for 5 days). Both onion and garlic treatments caused a temporary hypoglycemia as well as liver glycogen muscle depletion accompanied with a significant elevation in white muscle glycogen suggesting that dietary onion or garlic may enhance the process of glycogenesis in white muscle. Onion feeding caused hypolipidemia accompanied with significant decreases in liver content of total lipids in response to the repeated doses for 5 days and a severe rise in the lipid content of red muscle at all periods. Such results suggest that dietary onion may enhance the process of lipogenesis in red muscle of Clarias lazera. Probably, the main precursor of such lipogenesis in the red muscle was the serum fatty acids. Moreover, dietary onion led to hypocholesterolemia accompanied with a significant rise in cholesterol level of both liver and red muscle. On the other hand, dietary garlic significantly decreased serum free amino acids after 5 h and 5 days. Furthermore, significant elevations of liver free amino acids were dedected in the onion-treated fish group (24 h and 5 days) and in the garlic-treated fish group (5 h), probably due to oxidation of thiol components (sulfur attached to the proteins). This result was accompanied with either a marked increase in white muscle free amino acids at all periods of garlic fish group, or a noticeable drop in the onion group (repeated doses). Also, dietary onion and garlic treatment led to an elevation in hepatic ureagenesis in spite of the unchanged serum urea level. Significant decreases of both serum AST and ALT in garlic groups (all periods) and in onion groups (repeated doses) were observed. In liver, AST and ALT showed temporary rises after onion or garlic supplementation. In general, it could be concluded that both dietary onion and garlic have a transitory hypoglycemic action as well as a promoting effect on the process of glycogenesis in white muscle whereas dietary onion promotes lipogenesis in red muscle of fish. Also, dietary garlic may enhance the white muscle uptake of free amino acids which, in turn, may promote protein synthesis.

 

Just saying what it's used for-have never used it-now def won't. Thanks for the info

 

Responding to the original post: from my research and many proven studies, the only way to rid your tank of ich once introduced is to run the tank fishless for 6 to 9 weeks, depending on what you read. The life cycle of ich is impossible to determine, so the 9 week mark is the safest. Without a fish to host on, it will die. Then it is also a high possibility that the 2 new tangs you introduced already had ich. You cannot see ich with the naked eye, and they usually attack the gills and mouth first. That is why quarantine is highly suggested, before adding a fish to your display tank.

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god alot for me to read anyway.............

OMG i cant believe it woke up this morning to find.............. no ich on the yellow tang, i cant believe it fresh water dipping must work thanks PINK4MISS, im now a very happy marine keeper bubbles thanks you too

also my tank is quite small but my uv sterizler is 9w green mean killing machine

 

great to hear it worked for you, you might have to do it again in a day. but don't do it unless you see spots. the less stress the better. if you can find it pick up some selcon, its helps to boost the fishes immune system, its just vitamins from what i understand. but its a good thing to add to the tangs food. as stated by others the ich is still in the tank, you want to get the fishes immune system to fight it off.
most dip the fish and put it into a quarantine tank. but i put mine back into my display tank.
i did this method about a year ago, it worked for me.

 

Just because you see no spots (cysts) does not mean the ICH parasite is gone! It only means you no longer are seeing the eggs (cysts) of the ICH parasite. Which is most likely still under the scales, in the gills of the fish.

 

i no its still in the tank, but bubbles looks better and is not skratching anymore